![Show Menu](styles/mobile-menu.png)
![Page Background](./../common/page-substrates/page0003.png)
7
t h
E u r o p e a n C o n g r e s s o n
Obesity and
Eating Disorder
Obesity 2018
Journal of Obesity & Eating Disorders
ISSN 2471-8203
A p r i l 1 2 - 1 3 , 2 0 1 8
Am s t e r d a m , N e t h e r l a n d s
Page 23
N
onalcoholic fatty liver disease (NAFLD) is rapidly becoming one of the
most prominent causes of liver disease worldwide. The rising incidence
of NAFLD is linked to the obesity epidemic and the subsequent metabolic
disorders associated with it. Although obesity is a main risk factor for the
development of NAFLD, it can also arise in lean subjects at any age including
the pediatric age group. Non-obese fatty liver disease (NOFLD) can be
encountered in different clinical setting and in association with an array of
genetic, autoimmune, nutritional, drug-induced and metabolic, disorders
including lysosomal acid lipase deficiency. In this presentation, we will discuss
the general common features and the clinical conditions associated with
NAFLD and NOFLD.
Biography
H Hesham A-Kader is currently working as Professor at the
University Arizona. He received his MD degree from the Uni-
versity of Cairo. He completed his Masters in Pediatrics from
the University of Cairo. He then worked at the University of New
York served as Associate Professor and Professor at the Uni-
versity of Arizona. He has published several research papers
and original articles and chapters in prestigious journal and
major textbooks. His publications reflect his research interests
in Hepatology and fatty liver. He is certified by the Boards of Pe-
diatrics, Pediatric Gastroenterology and the Board of Nutrition.
He is also an editorial member and reviewer of several journals.
He is interested in the field of pediatric Hepatology specifically
in neonatal cholestasis and fatty liver disease,
hassan@peds.arizona.eduNAFLD and NOFLD: obese or slim it will get you
H Hesham A-Kader
The University of Arizona, USA
H Hesham A-Kader, J Obes Eat Disord 2018, Volume: 4
DOI: 10.21767/2471-8203-C1-008